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Behavior medication side effect




Pregunta

My daughter, age 21, began pacing after a second visit to the emergency room (ER) after the second bout with the flu. At that time, she was given Compazine for nausea. She was also given Phenergan during the visit. The pacing/walking movement never stopped since that time. Doctors after this visit, those in the hospital and in the psychiatric department, never addressed the pacing movement, even though they witnessed it. The neurologist did her homework and examined all the tests my daughter has had since February (CAT scan, EEG, etc) and all were normal.

She was administered two drugs which triggered this neurological disorder in my daughter-Compazine and Phenergan. These two drugs pressed a button in my daughter's brain and given constant doses of Haldol during her month-long stay in the psychiatric clinic only contributed to her disorder.

The neurologist recommended that my daughter never be given these two drugs ever again or Reglan, which is related to these drugs. She also said to make sure my daughter wears a tag that says she is allergic to these drugs and any other neuroleptic (consciousness altering) drugs. Unfortunately, we have to wait for the side effects to wear off. It will take months. In the mean time my daughter is on 1 mil of Cojentin to slow her down a little. All we can do is wait and hope this isn't a permanent disorder.



Respuesta de nuestros expertos

The problem is akathisia (neurological symptom of motor restlessness) and it is often drug-induced. Haldol and Compazine can cause this condition. It usually clears, though it may take months. Sometimes Klonopin or Inderal helps.



TG/TK 7-13-10

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Encuentre otras páginas que compartan el mismo tema que esta página El sistema nervioso autónomo7 El sistema nervioso autónomo21

 

Recomendación (es)

El sistema nervioso autónomo

R47
Las convulsiones en pacientes con SCdL deben tratarse utilizando los esquemas generales de tratamiento de la epilepsia.
R48
Solo se debe considerar la posibilidad de una resonancia magnética cerebral en el caso de que un paciente con SCdL muestre signos neurológicos aparte de una microcefalia (cabeza más pequeña de lo normal).

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